Skip to main navigation Skip to search Skip to main content

Simiao Decoction alleviates hyperuricemia-induced renal injury through regulating gut dysbiosis and decreasing gut-derived uremic toxins

  • Xinghong Zhou
  • , Xiaoyu Liu
  • , Baizhao Peng
  • , Ying Yang
  • , Hanqi Lu
  • , Dexian Li
  • , Yijian Deng
  • , Zihao Jiang
  • , Chuanghai Wu
  • , Wen Fang
  • , Yanting You
  • , Hiu Yee Kwan
  • , Xiaoshan Zhao*
  • , Yanyan Liu*
  • *Corresponding author for this work

Research output: Contribution to journalJournal articlepeer-review

2 Citations (Scopus)

Abstract

Renal injury is a common complication of hyperuricemia (HUA), which has been recognized as an independent risk factor for chronic kidney disease (CKD). The gut-kidney axis theory suggests that targeting the gut microbiota may be a potential treatment option for kidney disease. In this study, we utilized a spontaneous HUA rat model to demonstrate that Simiao decoction (SMD), a traditional Chinese medicine formula, can effectively alleviate HUA-induced renal injury by modulating gut microbiota and bacterial metabolism of tryptophan and tyrosine, thereby reducing gut-derived uremic toxins such as indoxyl sulfate (IS) and p-Cresol (PC). Fecal microbiota transplantation (FMT) further confirmed that the therapeutic effect of SMD was mediated by gut microbiota. Finally, in vitro studies revealed that IS promotes epithelial-mesenchymal transition (EMT) while PC induces cellular senescence in tubular cells. Collectively, our findings suggest that SMD can effectively alleviate HUA-induced renal injury through regulating gut dysbiosis and decreasing gut-derived uremic toxins. This study sheds light on a novel mechanism by which SMD exerts its effects on HUA-induced renal injury. (Figure presented.)

Original languageEnglish
Article number58
Number of pages19
Journalnpj Biofilms and Microbiomes
Volume12
Issue number1
DOIs
Publication statusPublished - 31 Jan 2026

Fingerprint

Dive into the research topics of 'Simiao Decoction alleviates hyperuricemia-induced renal injury through regulating gut dysbiosis and decreasing gut-derived uremic toxins'. Together they form a unique fingerprint.

Cite this