Metabolomics Reveal Nanoplastic-Induced Mitochondrial Damage in Human Liver and Lung Cells

Keyphrases

• Metabolomics 100%
• Mitochondrial Damage 100%
• Lung 100%
• Human Lung Cells 100%
• Human Liver Cells 100%
• Metabolic Function 100%
• L02 Cells 100%
• BEAS-2B Cells 100%
• Nanoplastics 100%
• Cell Death 50%
• Mitochondria 50%
• Human Health 50%
• Normal Human 50%
• Liver 50%
• Biosphere 50%
• Metabolic Pathway 50%
• Human Cells 50%
• Mitochondrial Membrane Potential 50%
• Untargeted Metabolomics 50%
• Mitochondrial Function 50%
• Mitochondrial Respiration 50%
• Mitochondrial Dysfunction 50%
• Overproduction 50%
• Adverse Outcomes 50%
• Environmental Levels 50%
• Functional Pathways 50%
• Toxicity Pathways 50%
• Mitochondrial Reactive Oxygen Species (mtROS) 50%
• Metabolic Toxicity 50%
• Plastic Debris 50%
• Transmission Electron Microscopy Analysis 50%

Pharmacology, Toxicology and Pharmaceutical Science

• Nanoplastics 100%
• Normal Human 50%
• Reactive Oxygen Metabolite 50%
• Transmission Electron Microscopy 50%
• Electron Microscopy 50%
• Adverse Outcome 50%