Integration of proteomics and metabolomics reveals promotion of proliferation by exposure of bisphenol S in human breast epithelial MCF-10A cells

Biochemistry, Genetics and Molecular Biology

• Proteomics 100%
• Epidermal Growth Factor Receptor 100%
• Metabolite 100%
• Metabolomics 100%
• Citric Acid Cycle 50%
• Signal Transduction 50%
• Mass Spectrometry 50%
• Cell Proliferation 50%
• Metabolic Pathway 50%
• Upregulation 50%
• Estrogen Receptor 50%
• Low Drug Dose 50%
• Lipid Metabolism 50%
• Purine Metabolism 50%
• Mammalian Target of Rapamycin 50%
• Quantitative Proteomics 50%
• Normal Human 50%

Keyphrases

• Proteomics 100%
• Bisphenol A 100%
• Metabolomics 100%
• Human Breast 100%
• MCF10A Cells 100%
• Epidermal Growth Factor Receptor 16%
• MCF10A 16%
• Tricarboxylic Acid Cycle (TCA cycle) 8%
• Lipid Metabolism 8%
• Normal Human 8%
• Epithelial Cell Line 8%
• Cell Proliferation 8%
• Quantitative Proteomics 8%
• Purine Metabolism 8%
• Mass Spectrometry-based Metabolomics 8%
• Endogenous Metabolites 8%
• Mammalian Target of Rapamycin (mTOR) 8%
• Induced Proliferation 8%
• Cell Signaling Pathways 8%
• Joint Pathway Analysis 8%
• CDH1 8%
• Pyruvate Metabolism 8%
• Endocrine Disrupting Effects 8%
• Protein Markers 8%
• Human Breast Epithelial Cells 8%
• MKI67 8%
• Normal Breast 8%
• Suggested Changes 8%
• Hormetic 8%
• Proliferation Rate 8%
• Estrogen Receptor-negative 8%
• Receptor-mediated 8%
• Breast Cell Lines 8%
• Estrogenic Effect 8%

Pharmacology, Toxicology and Pharmaceutical Science

• 4,4' Isopropylidenediphenol 100%
• Epidermal Growth Factor Receptor 100%
• Normal Human 50%
• Purine 50%
• Estrogen Receptor 50%
• Tricarboxylic Acid 50%
• Pyruvic Acid 50%