Press conference on the new regulatory mechanism of satiety and therapeutic target for obesity research

Press/Media

Description

A research team led by Hong Kong Baptist University (HKBU) has found that a proteolytic enzyme called membrane-type 1 matrix metalloproteinase (MT1-MMP) plays an important role in the regulatory mechanism of fullness, or satiety, and it could serve as a promising potential drug target for the management of obesity. The research findings were published in the internationally-renowned scientific journal Nature Metabolism. The study has also been featured as a research highlight in multiple high-impact journals, including Nature Reviews Endocrinology, Nature Metabolism and Science Signaling.

Period30 May 2022 → 31 May 2022

Media coverage

5

Media coverage

  • Title浸大團隊發現藥物可提升飽腹感 冀日後有助治療肥胖病人
    Degree of recognitionLocal
    Media name/outletRTHK News feature podcast
    Media typeRadio
    Country/TerritoryHong Kong
    Date31/05/22
    PersonsHoi Leong Xavier WONG
  • TitleHong Kong Today - New discovery could help weight loss
    Degree of recognitionLocal
    Media name/outletRTHK Radio 3
    Media typeRadio
    Country/TerritoryHong Kong
    Date30/05/22
    PersonsHoi Leong Xavier WONG
  • Title研究發現以藥物抑制一種蛋白酶可提升飽腹感從而減肥
    Degree of recognitionLocal
    Media name/outletRTHK
    Media typeWeb
    Country/TerritoryHong Kong
    Date30/05/22
    PersonsHoi Leong Xavier WONG
  • Title浸大發現一種蛋白水解酶可調節大腦發出飽腹感訊號 助研治療肥胖症藥物
    Degree of recognitionLocal
    Media name/outletTVB
    Media typeTelevision
    Country/TerritoryHong Kong
    Date30/05/22
    PersonsHoi Leong Xavier WONG
  • Title浸大研究發現抑制一種金屬蛋白酶可調節飽腹感
    Degree of recognitionLocal
    Media name/outletNow TV
    Media typeTelevision
    Country/TerritoryHong Kong
    Date30/05/22
    PersonsHoi Leong Xavier WONG

Media contributions

1

Media contributions

  • TitleHKBU-led research identifies new regulatory mechanism of satiety and therapeutic target for obesity
    Country/TerritoryHong Kong
    Date30/05/22
    PersonsHoi Leong Xavier WONG, Zhaoxiang BIAN